Clinical Education
AES is not a product supplier. It is a full clinical, diagnostic, training, and management ecosystem for dry eye care.
AES Diagnostic System
A structured 4-step diagnostic hierarchy ensuring comprehensive assessment from lids to tear film.
Lids & Lashes
Assess lid margin disease, blepharitis, demodex, and meibomian gland function.
Ocular Surface
Evaluate corneal and conjunctival staining, tear break-up time, and osmolarity.
Inflammation
Identify inflammatory markers, redness, and systemic contributors.
Functional Risk
Assess tear film stability, blink dynamics, and environmental factors.
The 4 Pillars of Dry Eye Management
Each pillar targets a specific aspect of dry eye disease for comprehensive treatment.
Eyelid Biofilm Control
Targets blepharitis, demodex, bacterial load and rosacea through in-office debridement, at-home eyelid cleansing gel, brush, HOCL spray and lid wipes.
Meibomian Gland Obstruction
Restores oil flow and blink efficiency through warm compresses, blink exercises, gland expression and thermal pulsation therapy.
Inflammation Control
Manages ocular surface, lid and systemic inflammation with topical steroids, cyclosporine, Omega-3 supplementation and lifestyle modifications.
Tear Film & Osmolarity
Optimises tear stability and hydration with type-specific artificial tears, punctal plugs, environmental control and proper hydration protocols.
3-Tier Management
Treatment progression from basic home care to advanced in-office procedures.
Entry / Home Care
At-home eyelid hygiene, warm compresses, HOCL spray, artificial tears, and Omega-3 supplementation. The foundation of every dry eye treatment plan.
Intermediate
In-office lid debridement, meibomian gland expression, anti-inflammatory drops (short-course steroids), and advanced tear supplements.
Advanced
Thermal pulsation therapy, cyclosporine/lifitegrast, punctal plugs, specialised procedures, and referral pathways for refractory cases.
Dry Eye Classification
Understanding the subtype guides treatment selection.
| Type | Key Markers | Primary Treatment | Secondary Treatment |
|---|---|---|---|
| Aqueous Deficient | Low Schirmer, low TMH, punctate staining | Tear supplementation, punctal plugs | Anti-inflammatory, secretagogues |
| Lipid Deficient (MGD) | Poor NIBUT, lid margin changes, meibography loss | Lid hygiene, gland expression, warm compress | Thermal pulsation, anti-inflammatory |
| Mucin Deficient | Goblet cell loss, chemical injury, Stevens-Johnson | Mucin-mimetic drops, autologous serum | Specialist referral |
| Mixed | Combination of above markers | Multi-pillar approach based on dominant subtype | Sequential treatment targeting each component |